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ApoE-Directed Therapeutics Rapidly Clear β-Amyloid and Reverse Deficits in AD Mouse Models

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Science  23 Mar 2012:
Vol. 335, Issue 6075, pp. 1503-1506
DOI: 10.1126/science.1217697

Article Information

vol. 335 no. 6075 1503-1506

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History: 
  • Received for publication December 9, 2011
  • Accepted for publication January 20, 2012

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Author Information

  1. Paige E. Cramer1,
  2. John R. Cirrito2,
  3. Daniel W. Wesson1,3,
  4. C. Y. Daniel Lee1,
  5. J. Colleen Karlo1,
  6. Adriana E. Zinn1,
  7. Brad T. Casali1,
  8. Jessica L. Restivo2,
  9. Whitney D. Goebel2,
  10. Michael J. James4,
  11. Kurt R. Brunden4,
  12. Donald A. Wilson3,
  13. Gary E. Landreth1,*
  1. 1Department of Neurosciences, Case Western Reserve University School of Medicine, Cleveland, OH 44106, USA.
  2. 2Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer’s Disease Research Center, Washington University School of Medicine, St. Louis, MO 63110, USA.
  3. 3Emotional Brain Institute, Nathan Kline Institute for Psychiatric Research and the New York University School of Medicine, Orangeburg, NY 10962, USA.
  4. 4Center of Neurodegenerative Disease Research, Department of Pathology and Laboratory Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104, USA.
  1. *To whom correspondence should be addressed. E-mail: gel2{at}case.edu

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