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Interleukin-3 amplifies acute inflammation and is a potential therapeutic target in sepsis

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Science  13 Mar 2015:
Vol. 347, Issue 6227, pp. 1260-1265
DOI: 10.1126/science.aaa4268

Article Information

vol. 347 no. 6227 1260-1265

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History: 
  • Received for publication December 4, 2014
  • Accepted for publication January 21, 2015

Author Information

  1. Georg F. Weber1,2,*,,
  2. Benjamin G. Chousterman1,*,
  3. Shun He1,*,
  4. Ashley M. Fenn1,
  5. Manfred Nairz1,
  6. Atsushi Anzai1,
  7. Thorsten Brenner3,
  8. Florian Uhle3,
  9. Yoshiko Iwamoto1,
  10. Clinton S. Robbins1,
  11. Lorette Noiret1,
  12. Sarah L. Maier2,
  13. Tina Zönnchen2,
  14. Nuh N. Rahbari2,
  15. Sebastian Schölch2,
  16. Anne Klotzsche-von Ameln4,
  17. Triantafyllos Chavakis4,
  18. Jürgen Weitz2,
  19. Stefan Hofer3,
  20. Markus A. Weigand3,
  21. Matthias Nahrendorf1,
  22. Ralph Weissleder1,5,
  23. Filip K. Swirski1,*,
  1. 1Center for Systems Biology, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.
  2. 2Department of Visceral, Thoracic and Vascular Surgery, Technische Universität Dresden, Dresden, Germany.
  3. 3Department of Anesthesiology, University of Heidelberg, Heidelberg, Germany.
  4. 4Department of Clinical Pathobiochemistry and Institute for Clinical Chemistry and Laboratory Medicine, Technische Universität Dresden, Dresden, Germany.
  5. 5Department of Systems Biology, Harvard Medical School, Boston, MA 02115, USA.
  1. Corresponding author. E-mail: fswirski{at}mgh.harvard.edu (F.K.S.); georg.weber{at}uniklinikum-dresden.de (G.F.W.)
  1. * These authors contributed equally to this work.

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