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Alternative polyadenylation of Pax3 controls muscle stem cell fate and muscle function

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Science  08 Nov 2019:
Vol. 366, Issue 6466, pp. 734-738
DOI: 10.1126/science.aax1694

Article Information

vol. 366 no. 6466 734-738

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History: 
  • Received for publication February 27, 2019
  • Accepted for publication September 26, 2019
  • .

Author Information

  1. Antoine de Morree1,2,*,
  2. Julian D. D. Klein1,2,
  3. Qiang Gan1,2,
  4. Jean Farup1,2,3,
  5. Andoni Urtasun1,2,
  6. Abhijnya Kanugovi1,2,
  7. Biter Bilen1,2,
  8. Cindy T. J. van Velthoven1,2,,
  9. Marco Quarta1,2,4,,
  10. Thomas A. Rando1,2,4,*
  1. 1Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.
  2. 2Paul F. Glenn Center for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.
  3. 3Departments of Clinical Medicine and Biomedicine, Research Laboratory for Biochemical Pathology, Aarhus University, Aarhus, Denmark.
  4. 4Center for Tissue Regeneration, Repair, and Restoration, Veterans Affairs Palo Alto Health Care System, Palo Alto, CA, USA.
  1. *Corresponding author. Email: demorree{at}stanford.edu (A.d.M.); rando{at}stanford.edu (T.A.R.)
  • Present address: Allen Institute for Brain Science, Seattle, WA, USA.

  • Present address: Molecular Medicine Research Institute, Sunnyvale, CA, USA.

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