Research Article

Noncanonical transnitrosylation network contributes to synapse loss in Alzheimer’s disease

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Science  15 Jan 2021:
Vol. 371, Issue 6526, eaaw0843
DOI: 10.1126/science.aaw0843

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vol. 371 no. 6526

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History: 
  • Received for publication November 15, 2018
  • Accepted for publication November 18, 2020
  • .

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Author Information

  1. Tomohiro Nakamura1,2,*,,
  2. Chang-ki Oh1,2,,
  3. Lujian Liao1,,
  4. Xu Zhang1,2,
  5. Kevin M. Lopez2,
  6. Daniel Gibbs3,
  7. Amanda K. Deal1,
  8. Henry R. Scott1,
  9. Brian Spencer3,
  10. Eliezer Masliah3,§,
  11. Robert A. Rissman3,4,
  12. John R. Yates III1,
  13. Stuart A. Lipton1,2,3,*
  1. 1Departments of Molecular Medicine and Neuroscience, and Neuroscience Translational Center, The Scripps Research Institute, La Jolla, CA 92037, USA.
  2. 2Neurodegenerative Disease Center, Scintillon Institute, San Diego, CA 92121, USA.
  3. 3Department of Neurosciences, University of California, San Diego, School of Medicine, La Jolla, CA 92093, USA.
  4. 4VA San Diego Healthcare System, San Diego, CA 92161, USA.
  1. *Corresponding author. Email: tnakamura{at}scripps.edu (T.N.); slipton{at}scripps.edu (S.A.L.)
  1. These authors contributed equally to this work.

  • Present address: Shanghai Key Laboratory of Regulatory Biology and Shanghai Key Laboratory of Brain Functional Genomics, School of Life Sciences, East China Normal University, Shanghai 200241, China.

  • § Present address: National Institute on Aging, NIH, Bethesda, MD 20892, USA.

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