Supplementary Materials

Host genetic diversity enables Ebola hemorrhagic fever pathogenesis and resistance

Angela L. Rasmussen, Atsushi Okumura, Martin T. Ferris, Richard Green, Friederike Feldmann, Sara M. Kelly, Dana P. Scott, David Safronetz, Elaine Haddock, Rachel LaCasse, Matthew J. Thomas, Pavel Sova, Victoria S. Carter, Jeffrey M. Weiss, Darla R. Miller, Ginger D. Shaw, Marcus J. Korth, Mark T. Heise, Ralph S. Baric, Fernando Pardo-Manuel de Villena, Heinz Feldmann, Michael G. Katze

Materials/Methods, Supplementary Text, Tables, Figures, and/or References

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  • Materials and Methods
  • Figs. S1 to S9
  • Captions for Tables S1 to S3
Table S1
Viral variants identified by RNAsequencing. Workbook tabs show sequence of viral RNA reads mapped to EBOV sequences specified by NCBI accession number.
Table S2
Significantly differentially expressed genes in spleen. Workbook tabs indicate conditions for lists of DEG from spleen (FDR-corrected p-value < 0.05, fold change >|1.5|)
Table S3
Significantly differentially expressed genes in liver. Workbook tabs indicate conditions for lists of DEG from liver (FDR-corrected p-value < 0.05, fold change >|1.5|)
Data File S1
Data File S2
Data File S3
Data File S4
Data File S5
Data File S6
Data File S7